Apoptosis Collection

Coloured SEM of lymphocytes attacking cancer cell
Cancer cell death. Coloured Scanning Electron Micrograph (SEM) showing killer T-lymphocytes (orange) inducing a cancer cell (mauve) to undergo Programmed Cell Death (PCD)

Carl Vogt, German naturalist
Carl Christoph Vogt (1817-1895), German naturalist. Vogt published a number of notable works on physiology, geology and zoology

Scanning electron micrograph of an apoptotic HeLa cell. Zeiss Merlin HR-SEM

Programmed cell death protein molecule F006 / 9597
Human programmed cell death protein 4, molecular model. This protein is involved in apoptosis (programmed cell death)

Programmed cell death protein 6 molecule F006 / 9548
Programmed cell death protein 6, molecular model. This is a calcium-binding protein is involved in apoptosis (programmed cell death)

Transcription factor and DNA molecule F006 / 9484
Transcription factor and DNA molecule. Molecular model of glucocorticoid receptor (GR) transcription factor protein (pink and blue) complexed with a molecule of DNA (deoxyribonucleic acid)

Caspase-3 and inhibitor F006 / 9457
Apopain protein and inhibitor. Molecular model of caspase-3, also known as apopain, complexed with an inhibitor. Caspase-3 is a protease, an enzyme that cleaves proteins

Caspase-9 with inhibitor, molecular model F006 / 9442
Caspase-9 complexed with an inhibitor, molecular model. Caspase-9 is a protease, an enzyme that cleaves proteins, that plays a role in apoptosis (programmed cell death)

RuvBL1 helicase enzyme, molecular model. Helicases are enzymes that carry out several roles, primarily separating the two strands of the DNA (deoxyribonucleic acid) double helix

Stem cell dying, SEM
Stem cell dying. Coloured scanning electron micrograph (SEM) of a stem cell undergoing apoptosis, or programmed cell death. Apoptosis occurs when a cell becomes old or damaged

Cell destruction, artwork
Cell destruction. Artwork of a cell undergoing destruction. This image could represent programmed cell death (apoptosis), a normal part of a cell life cycle

VDAC-1 ion channel protein C015 / 8250
VDAC-1 ion channel protein, molecular model. This is the human voltage-dependent anion-selective channel protein 1 (VDAC-1)

VDAC-1 ion channel protein C015 / 8249
VDAC-1 ion channel protein, molecular model. This is the human voltage-dependent anion-selective channel protein 1 (VDAC-1)

Torn muscle, illustration C018 / 0806
Torn muscle. Illustration of a tear in a skeletal muscle. Muscle tears can either result in muscle growth or injury depending on the circumstances

Carl Vogt, German naturalist C018 / 7033
Carl Vogt (1817-1895). 1869 engraving of the German naturalist Carl Christoph Vogt holding a skull. Vogt published a number of notable works on physiology, geology and zoology

Apoptosome cell death protein C015 / 6108
Apoptosome cell death protein, molecular model. Apoptosomes are large protein structures formed during programmed cell death (PCD, also called apoptosis)

Apoptosome cell death protein C015 / 6109
Apoptosome cell death protein, molecular model. Apoptosomes are large protein structures formed during programmed cell death (PCD, also called apoptosis)

HeLa cell dying, SEM C017 / 8304
HeLa cell dying, coloured scanning electron micrograph (SEM). The cell in the foreground is undergoing apoptosis, or programmed cell death. Apoptosis occurs when a cell becomes old or damaged

Caspase 6 enzyme C015 / 5700
Capase 6 enzyme, molecular model. Caspases are proteases, enzymes that cleave proteins and play a role in apoptosis (programmed cell death)

Caspase 6 enzyme C015 / 5699
Capase 6 enzyme, molecular model. Caspases are proteases, enzymes that cleave proteins and play a role in apoptosis (programmed cell death)

Transcription factor and DNA molecule C015 / 5344
Transcription factor and DNA molecule. Molecular model of glucocorticoid receptor (GR) transcription factor protein (blue and purple) complexed with a molecule of DNA (deoxyribonucleic acid)

Transcription factor and DNA molecule C015 / 5343
Transcription factor and DNA molecule. Molecular model of glucocorticoid receptor (GR) transcription factor protein (yellow and green) complexed with a molecule of DNA (deoxyribonucleic acid)

Human tumour protein p73 C016 / 2830
Human tumour protein p73. Molecular model of the human tumour protein p73, encoded by the TP73 gene. Like the similar p53, is is a transcription factor of a type known as a tumour suppressor gene

Human tumour protein p73 C016 / 2831
Human tumour protein p73. Molecular model of the human tumour protein p73, encoded by the TP73 gene. Like the similar p53, is is a transcription factor of a type known as a tumour suppressor gene

Apoptosis, conceptual image C013 / 9998
Apoptosis, conceptual image. Computer artwork representing apoptotic cells. Apoptosis is the process of programmed cell death

Apoptosis, conceptual image C013 / 9997
Apoptosis, conceptual image. Computer artwork representing apoptotic cells. Apoptosis is the process of programmed cell death

Coloured SEM of lymphocyte attacking cancer cell
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Carl Vogt, German naturalist
Carl Christoph Vogt (1817-1895), German naturalist. Vogt published a number of notable works on physiology, geology and zoology

Programmed cell death. Comparison of a fluorescent light micrograph (top) and transmission electron micrograph (TEM, bottom) of programmed cell death (apoptosis)

Cell death. Computer-enhanced confocal light micrograph of cells in the retina of the eye undergoing programmed cell death (apoptosis)

Immunfluorescent LM of cell death (apoptosis)

Cellular clock. Conceptual artwork of a clock and the internal structures (organelles) of an animal cell. This could represent the rate of metabolic cell reactions

Intestinal villus cell loss, SEM
Intestinal villus cell loss, coloured scanning electron micrograph (SEM). Close-up of the tip of a villus in the small intestine that has shed a cell from its tip as part of the normal cell

Intestinal villi cell loss, SEM
Intestinal villi cell loss, coloured scanning electron micrograph (SEM). Close-up of the tips of villi in the small intestine

Apoptosis, fluorescent light micrograph
Apoptosis. 2-photon fluorescence light micrograph of a cultured cell (centre) undergoing apoptosis, or programmed cell death. It is surrounded by healthy cells

Caspase 3 molecule
Caspase-3 molecule. Computer artwork showing the secondary structure of a molecule of caspase-3. Caspase-3 is a protease, an enzyme that cleaves proteins

Caspase 1 molecule
Caspase-1 molecule. Computer artwork showing the secondary structure of a molecule of caspase-1. Caspase-1 is a protease, an enzyme that cleaves proteins

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Apoptosis, the programmed cell death mechanism, is a captivating phenomenon that holds immense significance in various fields of research. This coloured scanning electron micrograph showcases lymphocytes launching an attack on a cancer cell, illustrating the intricate process at work. First discovered by Carl Vogt, a renowned German naturalist, it has since become an area of intense scientific exploration. The groundbreaking R&D 100 Award Winning Mitogenesis-Coupled Apoptosis Molecular Device (MCAMD) Team has made significant strides in unraveling the complexities surrounding this cellular event. Through their meticulous efforts and innovative approach, they have shed light on key players involved in apoptosis. The microscopic image of an apoptotic HeLa cell provides us with a glimpse into the structural changes occurring during this process. Additionally, we can observe Programmed Cell Death Protein molecules F006/9597 and F006/9548 as well as Transcription Factor and DNA molecule F006/9484 actively participating in orchestrating apoptosis. Caspase-3 and its inhibitor F006/9457 play crucial roles in regulating this intricate dance of life and death within cells. Similarly, Caspase-9 with its inhibitor depicted through molecular model F006/9442 adds another layer to our understanding of how these proteins function together harmoniously. Moreover, the presence of RuvBL1 helicase enzyme further emphasizes the complexity behind apoptosis regulation. Its involvement highlights yet another facet requiring extensive investigation to comprehend fully. Thanks to pioneers like Carl Vogt and cutting-edge researchers such as the MCAMD team who continue pushing boundaries within this field; we are gradually uncovering the mysteries surrounding apoptosis. These captivating images provide glimpses into a world where cells decide their own fate – whether to survive or undergo programmed self-destruction for the greater good.