Budding HIV particle, computer artwork

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Budding HIV particle, computer artwork

Budding HIV particle. Computer artwork of an HIV (human immunodeficiency virus) particle (green) exiting a T-lymphocyte (a type of white blood cell, lower left). HIV causes AIDS (acquired immune deficiency syndrome). The virus consists of an RNA (ribonucleic acid) genome, surrounded by an icosahedral (20-sided) protein nucleocapsid (coat). The whole virus is enclosed in an envelope derived from the host cells plasma membrane. The envelope has viral proteins integrated into it, including glycoprotein spikes that aid its attachment to host cells. When the virus particles leave the host cell they kill it, leading to a very weak immune system

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Media ID 6412806

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Acquired Immune Deficiency Budding Enveloped Exiting Human Immunodeficiency Virus Icosahedral Immunology Infect Infected Infection Leaving Leucocyte Leucocytes Leukocyte Leukocytes Lymphocyte Lymphocytes Particle Particles Pathogenic Pathogens Protein Spike Retro Virus Rna Virus Spikes Syndrome T Lymphocyte Viral Virion Virions Virology Viruses White Blood Cell White Blood Cells Condition Disorder Micro Biology Microbiological Pathogen Virus

EDITORS COMMENTS
This computer artwork showcases the intricate structure of a budding HIV particle, shedding light on the complex nature of this devastating virus. The vibrant green hue represents the viral particle as it emerges from a T-lymphocyte, one of our body's defense mechanisms against infections. HIV, short for human immunodeficiency virus, is responsible for causing AIDS (acquired immune deficiency syndrome), a condition that weakens the immune system and leaves individuals vulnerable to various infections. This visual representation highlights key components of the virus: an RNA genome enclosed within an icosahedral protein nucleocapsid coat. The envelope surrounding the entire virus originates from the host cell's plasma membrane and contains integrated viral proteins, including glycoprotein spikes crucial for attaching to host cells during infection. Tragically, when these particles exit their host cell after replication, they destroy it in their wake—contributing to further weakening of the already compromised immune system. Through this stunning illustration, we gain insight into both biology and medicine by exploring how pathogens like HIV interact with our bodies at a microscopic level. It serves as a reminder of ongoing research efforts aimed at understanding and combating such viruses while offering hope for future advancements in immunology and virology fields.

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